The ketogenic diet places an emphasis on fat. It still allows for protein and carbohydrates, albeit in moderate to low amounts only. The ketogenic diet works by flipping your body’s metabolic switch from using glucose for energy to using fats and ketones. This can lead to many positive health outcomes, including weight loss.

Unfortunately, the ketogenic diet is tough to stick with. For starters, carbohydrates are normally found in foods that are staples in many cuisines, such as rice and bread. The ketogenic diet throws these foods out the window because of their high-carb content, leaving millions without their staples, as previously noted. Here Winnie Martin, Food.news, reflects on ketones addressing brain health and Alzheimer’s disease:

“Ketones should be a familiar term for those people undergoing a ketogenic diet. These multi-chain fatty acids are the result of the body breaking down fats to use for energy instead of carbohydrates. People on a ketogenic diet eat high amounts of fat and very low amounts of carbohydrates to enter a state of ketosis. Following a ketogenic diet causes weight loss and suppresses appetite, as the body uses fat for energy.

Aside from helping you lose weight, ketones produced by ketosis also contribute to brain health. In fact, a number of studies have proposed the ketogenic diet as an alternative therapy if you’re experiencing epileptic seizures. Two studies done in 1998 and in 2008 suggested that the ketogenic diet diminished the frequency of seizures in epileptic children.

A study performed in 2019 has also found that ketones also exhibit protective properties against the damage caused by Alzheimer’s disease. This degenerative brain disorder damaged neurons responsible for transmitting messages from the brain to other organs. Researchers behind the study discovered that a diet supplemented with ketones may help protect neurons from dying during the progress of Alzheimer’s disease.

In the early stages of the disease, the brain becomes over-excited due to the diminished number of inhibitory interneurons. These prevent other nerve cells from signaling too much – at the cost of requiring more energy. Interneurons are more susceptible to dying when they encounter the protein amyloid beta, which is produced by those suffering from Alzheimer’s disease. Amyloid beta has been shown to damage nerve cell mitochondria by interfering with SIRT3, a protein that protects the neurons and preserves mitochondrial functions.”

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